Question

a 55 year old male with a history of chronic NSAID use is diagnosed with a duodenal ulcer. what is the most likely underlying mechanism for ulcer formation in this patient? a. excess histamine release from enterochromaffin-like cells b. activation of vagal pathways increasing gastric motility c. reduced prostaglandin synthesis leading to direct mucosal injury from NSAIDs d. increased bicarbonate secretion causing mucosal irritation

Name: a 55 year old male with a history of chronic nsaid use is diagnosed with a duodenal ulcer what is the most likely underlying mechanism for ulcer formation in this patient a excess histamine 15357
Uploaded: 2021-11-19T23:33:10-08:00
Duration: 2 min 6 s
Channel: Joanna Quigley
Description: a 55 year old male with a history of chronic nsaid use is diagnosed with a duodenal ulcer what is the most likely underlying mechanism for ulcer formation in this patient a excess histamine 15357

          a 55 year old male with a history of chronic NSAID use is diagnosed with a duodenal ulcer. what is the most likely underlying mechanism for ulcer formation in this patient?
a. excess histamine release from enterochromaffin-like cells
b. activation of vagal pathways increasing gastric motility
c. reduced prostaglandin synthesis leading to direct mucosal injury from NSAIDs
d. increased bicarbonate secretion causing mucosal irritation

Added by Teresa M.

Biology for AP Courses

Julianne Zedalis, John Eggebrecht

Instant Answer

Solved by Expert Joanna Quigley

11/19/2021

Step 1

The patient is a 55-year-old male with a history of chronic NSAID (nonsteroidal anti-inflammatory drug) use and has been diagnosed with a duodenal ulcer. Show more…

Show all steps

Thanks for your feedback!

a 55 year old male with a history of chronic NSAID use is diagnosed with a duodenal ulcer. what is the most likely underlying mechanism for ulcer formation in this patient? a. excess histamine release from enterochromaffin-like cells b. activation of vagal pathways increasing gastric motility c. reduced prostaglandin synthesis leading to direct mucosal injury from NSAIDs d. increased bicarbonate secretion causing mucosal irritation