A researcher wanted to design a muscle that would contract quickly, but relax slowly. Which mutation listed below would cause the muscle to relax more slowly? Fast myosin heavy chain isoform More sarcoplasmic reticulum Increased abundance of voltage gated calcium channels Reduced acetylcholinesterase activity
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Step 1: The relaxation of a muscle is primarily controlled by the reuptake of calcium ions by the sarcoplasmic reticulum and the breakdown of acetylcholine by acetylcholinesterase. Show more…
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Imagine the troponin in a skeletal muscle fiber had a mutation that prevented its binding to calcium but allowed it to bind to tropomyosin. Which of the following would increase the amount of force generated by the muscle fiber? Assume ATP levels are normal. Choose every correct answer. Group of answer choices: Inhibiting calcium release from the sarcoplasmic reticulum, Opening voltage-gated sodium channels, Acetylcholine receptor stimulation such that the fiber reaches threshold, Opening voltage-sensitive calcium channels, Nothing would cause an increase in force generated by the muscle fiber.
Kaela P.
A patient shows impaired smooth muscle relaxation but no effects of cardiac or skeletal muscle relaxations. The electrolyte levels are normal. Which of the following statements is the best explanation for this defect? Select one: a. The patient has a mutation in their myosin light chain phosphatase which is impairing function. b. The patient has a mutation in their myosin light chain kinase which is impairing function. c. The patient has a mutation in their L-type Ca2+ channel which is impairing function d. The patient has a mutation in their troponin which is impairing function
Adi S.
Smooth muscle, in contrast with skeletal muscle, is not regulated by a tropomyosin-troponin mechanism. Instead, vertebrate smooth-muscle contraction is controlled by the degree of phosphorylation of its light chains. Phosphorylation induces contraction, and dephosphorylation leads to relaxation. Like that of skeletal muscle, smooth-muscle contraction is triggered by an increase in the cytoplasmic calcium ion level. Propose a mechanism for this action of calcium ion on the basis of your knowledge of other signal-transduction processes.
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