Part A Certain drugs used to treat dysrhythmias, called local anesthetics, work by blocking voltage gated sodium ion channels and slowing the entry of sodium ions into the cell during an action potential. What effects would you see? Increase in hyperpolarization Decrease repolarization of cell Increase depolarization of the cell Decrease depolarization of the cell
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Potassium channel blockers, such as amiodarone and procainamide, which are used to treat abnormal electrical activity in the heart, impede the movement of K+through voltage-gated K+channels. Which part of the action potential would potassium channels affect, and why? a. Depolarization after peak action potential would be affected because that is the point when K+ begins to leave the cell. b. Repolarization after peak action potential would be affected because that is the point when K+ begins to leave the cell. c. Repolarization after peak action potential would be affected because that is the point when K+ begins to enter the cell. d. Polarization after peak action potential would be affected because that is the point when K+ begins to enter the cell.
Celine I.
Figure 35.11 Potassium channel blockers, such as amiodarone and procainamide, which are used to treat abnormal electrical activity in the heart, called cardiac dysrhythmia, impede the movement of $K+$ through voltage-gated $\mathrm{K}+$ channels. Which part of the action potential would you expect potassium channels to affect?
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