rounded, eosinophilic droplets, vacuoles, oraggregates in the cytoplasm causes: reabsorption droplets in proximal renal tubules —> proteinuria
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Acute glomerulonephritis (GN) is sudden inflammation of the glomeruli, most commonly a result of a streptococcal infection that causes the body to attack its own tissue. The damage to the filtration membrane increases permeability of the membrane for proteins and larger components. Describe the abnormal components of the urine for patients with GN and how these changes in filtrate will impact Tubular Reabsorption.
Adi S.
Below are fluorescent micrographs (from indirect fluorescence of the renal tubule). What can you conclude by analyzing these results? Control + OH-Pro + Glyoxylate CALB1 - protein found on the surface of collecting duct epithelial cells LTL - lectin binding proteins found enriched on proximal convoluted tubules DAPI - nuclear stain (not antibody) that upon entry into cells stains blue (used as control to enable us to see the cortical and medullary tissue) Select one: a. The collecting ducts of glyoxalate-treated but not hydroxyproline-treated (OH-pro) rats have a greater diameter. b. The same secondary antibody is used to detect CALB1 and LTL proteins c. Rats with calcium oxalate crystals enlarge the diameter of both their proximal convoluted tubule and collecting duct renal tubules d. The different primary antibodies used in this experiment detect the mRNA for the CALB1 and LTL genes, respectively. e. C and D are correct
Why is protein in the urine a sign of kidney damage? What structures in the kidney are probably affected?
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