The contractile cardiac muscle action potential is: 1- unique due to its extended hyperpolarized state 2- similar to autorhythmic cardiac muscle action potentials 3- similar to skeletal muscle action potentials 4- unique due to its voltage-gated Cl- channels 5- unique due to its extended plateau phase
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Which of the following statements about the action potentials in contractile myocardial cells (cardiac muscle) is INCORRECT? Answers: 1. Compared to skeletal muscle cells, contractile myocardial cells have a lower (more negative) resting membrane potential. 2. While cells in the SA node are autorhythmic and are able to generate action potentials without any outside stimuli, contractile myocardial cells require an outside stimulus to generate an action potential. 3. The duration of an action potential in a contractile myocardial cell is greater than the duration of an action potential in a typical neuron. 4. During the repolarization phase of a contractile myocardial action potential, the cell is highly permeable to sodium, potassium, and calcium.
Rupsa S.
The plateau phase of an action potential in cardiac muscle cells is represented in the figure, and it is due to the following: - Tension development (contraction) - Membrane potential (mV) - Absolute refractory period - Time (ms) - Efflux of K+ through K+ channels - Influx of Na+ through fast Na+ channels - Influx of Ca2+ through slow Ca2+ channels - Efflux of Ca2+ through fast Ca2+ channels - Efflux of Na+ through fast Na+ channels
Madhur L.
Put the steps of muscle contraction in order. Start with the arrival of the Action Potential (AP) arriving at the neuromuscular junction. Depolarization of the sarcolemma (plasma membrane) occurs. Repolarization (relaxation) occurs. Action potential from the nerve arrives at the Neuromuscular Junction. Myosin binds to exposed sites on actin, & pulls the actin towards the center of the sarcomere (contraction). Acetylcholine diffuses across the synaptic cleft and binds to receptors on the Sarcolemma. Action potential propagates down the t-tubules & causes the release of calcium ions (Ca2+), which binds to the troponin-tropomyosin complex which exposes the binding sites on actin. After contraction, ATP causes the myosin/actin connection to break.
Suman K.
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