various approaches in dealing with parasitic infections (Both Protozoan and Helminth) in immunocompromised individuals. Be sure to include prophylaxis and suppressive therapy in your discussions as well as the types of antiparasitic agents used?
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Sri K.
1. What are the infective stages of the giant roundworm and the whipworm to humans? 2. What is the age group most commonly affected by Ascariasis and Trichuriasis? Research on this and provide a brief explanation. 3. Explain the pathogenesis of Hookworm infection in man by making an outline of its chain of infection (provide the appropriate 6 elements). 4. What is the infective stage of Plasmodium falciparum in man? 5. In the life cycle of Plasmodium ovale and Plasmodium vivax, what developmental stage causes episodes of relapses in the human phase? 6. Briefly explain erythrocyte loss and jaundice of a patient with a high level of Malarial parasitism.
The intravenous administration of gamma globulin frequently improves autoimmune disease. It has been observed that the administration of gamma globulin to patients with pemphigus vulgaris significantly lowers the titer of IgG4 anti-desmoglein-3 and improves the clinical course of the disease. How do you explain this effect? Relatives of patients with pemphigus vulgaris who share the DRB1*0402 MHC class II type have IgG1 antibodies against desmoglein-3. These antibodies fix complement but do not cause disease. Can you speculate why this antibody does not cause disease, whereas the IgG4 antibody causes disease? What might explain the difference between unaffected people who make only the IgG1 antibody against desmoglein-3 and the patients who make the IgG4 antibody? Can you design an experiment to test your hypothesis? We have seen cyclophosphamide used to eradicate bone marrow cells in cases 8 and 27. Why is it useful? Why did Mr. Sammlier develop pneumonia caused by pneumocystis jirovecii during his initial treatment for pemphigus vulgaris?
Adi S.
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