With the release of a neurotransmitter, such as acetylcholine, into the synapse, an electrical signal has been converted to a chemical signal. What is the effect on the post-synaptic cell if the acetylcholine receptor is a ligand-gated sodium channel? The effect of acetylcholine is short-lived, given two mechanisms for the removal of the neurotransmitter from the synapse. What are these two mechanisms?