A child with the genes for PKU (a recessive disorder that leads to brain damage because proteins aren't digested properly) is raised to adulthood under a strict low-protein diet that prevents the gene from being activated. He lives a normal life and eventually becomes a father. As a genetic counselor, explain whether the fact that his gene is inactive affects the probability his future children might inherit the PKU allele. Be complete. Explain the reasoning you used to reach your conclusion.
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In humans, there is a disease called Phenylketonuria (PKU) which is caused by a recessive allele. People with this allele have a defective enzyme and cannot break down the amino acid phenylalanine. This disease can result in mental retardation or death. Let "E" represent the normal enzyme. Also, in humans, there is a condition called galactose intolerance or galactosemia, which is also caused by a recessive allele. Let "G" represent the normal allele for galactose digestion. In both diseases, normal dominates over recessive. If two adults were heterozygous for both traits (EeGg), what are the chances of having a child that is completely normal? Has just PKU? Has just galactosemia? Has both diseases?
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