Which of the following are NOT ideal antibacterial drug targets? (select ALL that apply) a. Peptidoglycan b. 60S ribosome c. Cell membrane d. DNA/RNA e. 50S ribosome f. Chitin g. Folic acid synthesis Which of the following antibiotics target bacterial cell walls? (select all that apply) a. Penicillin b. Tetracycline c. Macrolide d. Cephalosporin e. Metronidazole f. Vancomycin g. Trimethoprim h. Chloramphenicol i. Rifampin Which of the following antibiotics target bacterial protein synthesis? (select all that apply) a. Penicillin b. Tetracycline c. Macrolide d. Cephalosporin e. Metronidazole f. Vancomycin g. Trimethoprim h. Chloramphenicol i. Rifampin
Added by Michael B.
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a. Peptidoglycan - This is a component of bacterial cell walls and is not present in human cells, making it an ideal target. b. 60S ribosome - This is a component of eukaryotic (including human) ribosomes, not bacterial ribosomes, so it is NOT an ideal target. c. Show more…
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Antimicrobial Drug Mode of action Penicillin Affects peptidoglycan to cause cell wall damage and lysis Chloramphenicol Inhibits protein synthesis by binding to 70S ribosomes Binds to ergosterol to affect cell membrane Amphotericin B Quinines Causes buildup of toxic free radicals in parasite's cells Oseltamivir Prevents viral budding Refer to the table given above to answer the following: A) Which of the drugs given in the table above could be used to treat yeast infection with selective toxicity? B) Explain why your choice of antimicrobials would be selectively toxic: C) Explain why you eliminated all the other choices of antimicrobials.
Madhur L.
Match each of the following antibacterial drugs with its mechanism of action (can be used more than once): Antibacterial drugs: penicillins, sulfa drugs, bacitracin, beta-lactamases, aminoglycosides, cephalosporins Mechanism of action: a. prevent protein synthesis by binding to the 30S subunit b. inhibit folic acid synthesis by competing for the enzyme that carries out this process c. break the beta-lactam ring, which inactivates the beta-lactam drugs d. inhibits the peptidoglycan synthesis by preventing the dephosphorylation of bacterioprenol, which is the carrier molecule e. inhibits the peptidoglycan synthesis by binding to the bacterial transpeptidase
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